ecg_patterns
Table of Contents
patterns of ECGs
see also:
Obvious arrhythmias:
- idioventricular rhythm - complete heart block / atrial standstill
-
- bigeminy
- trigeminy
- couplets
- sinus rhythm
Bundle branch block or paced rhythm which may make interpretation of ischaemic changes difficult:
Hemiblocks:
- see also Fascicular blocks
LAHB:
- ECG criteria:
- LAD of -45 to -90deg
- small q wave 1-4mm in lead I
- small r wave 1-4mm in lead III
- normal QRS provided RBBB absent
- aetiology:
- IHD
- chronic LVF
- cardiomyopathy
- Chaga's disease
- children with endocardial cushion defect or tricuspid atresia
LPHB:
- uncommon as is short, thick with dual blood supply thus avoid making Dx
- ECG criteria (inaccurate though!):
- small r wave (<5mm) lead I & small q wave in leads III, aVF
- normal QRS duration
- exclude RVH and cor pulmonale
Acute myocardial infarct or ischaemia
Specific drug effects:
digoxin effect:
- reversed tick: ST depression & T inv. in V5-6
- toxic effects:
- any arrythmia esp. ventricular ectopy, nodal bradycardia
- NOT rapid AF
tricyclic OD:
- sinus tachycardia
- wide QRS
- prolonged QTc
- VT/VF
heart block:
- digoxin
- beta blockers
- calcium channel blockers
- lithium (sinus arrest rather than AV block)
wide QRS with prolonged QTc (although maybe normal):
- V-W class Ia drugs
- V-W class III drugs
- antipsychotic overdoses
- see prolonged QTc
wide QRS with normal QTc:
- flecainide
- moricizine
- phenytoin (but not lignocaine which has no effect on either)
- propranolol
Electrolyte disturbances:
-
- ⇒ tall tented T waves, widening QRS, loss of P wave, etc and eventually sinusoidal appearance, VF or asystole
-
- ⇒ ST depression, T wave flattening, prominent U wave creating a prolonged QTc effect, torsade de pointes VT
- ie. similar ECG effects as has lithium toxicity, although lithium toxicity is also characterised by sinus node dysfunction causing bradycardia, sinus arrest with junctional escape rhythm or asystole, and unlike hypokalaemia, ventricular arrhthmias are rare.
-
- ⇒ shortened QTc
-
- ⇒ prolonged QTc, torsade de pointes VT
Pericarditis:
- see also pericarditis for the classic four stages of ECG changes
- NB. tachycardia may be the only ECG finding if ST elevation has resolved & T waves remain normal
benign early repolarisation
- a cause of ST elevation in V1-4
LVH:
- tall R waves laterally +/- hyperacute T waves or LV strain pattern of ST depression with T inversion
- S waves in V1-2
RVH:
cardiomyopathy
dilated cardiomyopathy
- left atrial abnormality, eg. prolonged PR
- intraventricular conduction abnormalities esp. Left Bundle Branch Block (LBBB)
- poor precordial R wave progression
- ventricular arrhythmias
hypertrophic cardiomyopathy
- septal Q waves but upright T waves
- diminished or absent R waves in lateral leads
restrictive cardiomyopathy
- low voltage
- ventricular arrhythmias
- AV block
- complete heart block (sarcoidosis)
- acute coronary syndrome patterns (sarcoidosis)
dysrhythmogenic RV cardiomyopathy
Pulmonary embolus:
- see also pulmonary embolism (PE)
- non-specific right atrial strain patterns but these are often not present
- no significant abnormality in up to 25% of sub-massive PEs
- sinus tachycardia common if acute
- SI, QIII, TinvIII but not sens. and not spec.
- strain pattern V1-3 - symmetrical T wave inversion
- ST depression I or II
- SI, SII and SIII pattern
- Q waves in III & aVF
- Qr in V1
- ST elevation in V1, aVR & III
- RAD
- RBBB
- AF or atrial flutter
pre-excitation syndromes:
WPW:
- shortened PR interval
- delta wave
- dominant R in V1
- wide or narrow complex SVTs
ecg_patterns.txt · Last modified: 2024/09/08 03:25 by gary1